NATURE AND THE NATURE RESEARCH JOURNALS PRESS RELEASE
For papers that will be published online on 17 January 2010
This press release is copyrighted to the Nature journals mentioned below.
This press release contains:
· Summaries of newsworthy papers:
Cell Biology: Origin of fat build up in muscle
Genetics: A new risk locus for major mood disorders
Geoscience: Long-term contamination in Alaska
Genetics: New variants influence type 2 diabetes risk and blood sugar levels
Nature: Host factors implicated in influenza virus replication
Chemical Biology: Finding psychiatric drugs in zebrafish?
Geoscience: Indonesian earthquake risk
Methods: Assembling the bigger picture
And finally…Physics: ‘Chaos control’ spawns complex robotic behaviour
· Mention of papers to be published at the same time with the same embargo
· Geographical listing of authors
PDFs of all the papers mentioned on this release can be found in the relevant journal’s section of http://press.nature.com. Press contacts for the Nature journals are listed at the end of this release.
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PLEASE CITE THE SPECIFIC NATURE JOURNAL AND WEBSITE AS THE SOURCE OF THE FOLLOWING ITEMS. IF PUBLISHING ONLINE, PLEASE CARRY A HYPERLINK TO THE APPROPRIATE JOURNAL’S WEBSITE.
[1] & [2] Cell Biology: Origin of fat build up in muscle
DOI: 10.1038/ncb2014
DOI: 10.1038/ncb2015
An insight into the build up of fatty tissue in damaged muscle is published online in two papers in Nature Cell Biology this week. This fat deposition is associated with disorders such as muscular dystrophy and the research may have therapeutic relevance for future treatments into such disorders.
Healthy muscle regeneration is dependent on responses of multiple cell types, including satellite cells – known as adult muscle stem cells. Akiyoshi Uezumi and colleagues identify a population of progenitors, distinct from satellite cells, which are responsible for the formation of fatty tissue both in cell culture and in mice. These cells – PDGFRalpha+ – are inhibited by healthy muscle cells, but proliferate when transplanted into mouse damaged muscles.
In a related paper Fabio Rossi and colleagues also identify a population of fat forming progenitors that reside in muscle tissue. Using transplantation they show that these progenitors generate fatty tissue when delivered into damaged, but not healthy muscle in mice. These cells are not directly involved in repair, but can stimulate restoration of muscle function.
The researchers believe that targeting these fat progenitors may open new opportunities for designing therapeutic strategies to reducing scarring and treat muscle diseases.
Author contacts:
Akiyoshi Uezumi (Fujita Health University, Aichi, Japan) Author paper [1]
Tel +81 562 939 393; E-mail: [email protected]
Fabio Rossi (University of British Columbia, Vancouver, Canada) Author paper [2]
Tel +604 8227138; E-mail: [email protected]
[3] Genetics: A new risk locus for major mood disorders
DOI: 10.1038/ng.523
A genetic variant at chromosome 3p21.1 influences the risk of developing major mood disorders, including bipolar disorder and major depressive disorder. The discovery, online this week in Nature Genetics, supports the theory that the genetic variants that influence bipolar disorder and major depressive disorder overlap.
Bipolar disorder and major depressive disorder are two broad types of mood disorder, which together affect several hundred million individuals worldwide. Major depressive disorder is characterized by feelings of sadness and low self-worth, loss of pleasure, disturbed sleep and appetite, low energy, and poor concentration. Bipolar disorder is characterized by alternating periods of depression and elevated mood (mania). Manic behaviour often includes delusions of grandeur, hyperactivity, increased energy, and reckless behaviour. Both bipolar disorder and depression are associated with a high risk of suicide.
Francis McMahon and colleagues perform a meta-analysis of nearly 7,000 individuals with a major mood disorder and find that genetic variants at chromosome 3p21 were associated with increased risk of major mood disorders.
Author contact:
Francis McMahon (National Institutes of Health, Bethesda, MD, USA)
Tel: +1 301 451 4455; E-mail: [email protected]
[4] Geoscience: Long-term contamination in Alaska
DOI: 10.1038/ngeo749
Oil spilt from the tanker Exxon Valdez in 1989 can still be found on the Alaskan coast because of the two-layered structure of local beaches, according a study online this week in Nature Geoscience. As oil exploitation and shipping in the Arctic region becomes more feasible owing to global warming, effective environmental protection and clean-up of spilt oil will become increasingly important.
Michel Boufadel and Hailong Li investigated the groundwater dynamics of a beach on Eleanor Island, Alaska, which was contaminated by the Exxon Valdez oil spill, using field measurements, tracers and numerical simulations. They found that the upper layer of the beach acted as a reservoir for the spilt oil, protecting it from weathering and loss of fluidity. From this reservoir, the oil entered the lower layer whenever the water level fell below the interface between the two layers. Because of the low oxygen content in the lower layer, the oil is not degraded and can persist in the long term.
Author contact:
Michel Boufadel (Temple University, Philadelphia, PA, USA)
Tel: +1 215 204 7871; E-mail: [email protected]
[5] & [6] Genetics: New variants influence type 2 diabetes risk and blood sugar levels
DOI: 10.1038/ng.520
DOI: 10.1038/ng.521
Common genetic variants that are associated with risk of type 2 diabetes and blood glucose levels are reported in 2 studies online this week in Nature Genetics. Together, the reports shed light on the genetic variants that influence circulating glucose levels in the body, many of which also influence risk of type 2 diabetes.
More than 220 million people have diabetes worldwide, and 90% of those affected have type 2 diabetes (T2D). Clinically, T2D is defined by a chronic state of elevated blood sugar levels, when measured during fasting or 2 hours after an oral glucose challenge. The oral glucose challenge test is commonly performed to measure the body’s ability to use a defined amount of sugar. Typically, the patient is asked to ingest a sweet liquid containing glucose and blood samples are taken prior and after drinking the glucose. In T2D, the body does not produce enough insulin or the body’s cells cannot utilize insulin to properly manage blood sugar levels.
The MAGIC (Meta-Analyses of Glucose and Insulin-related traits Consortium) investigators led by Jose Florez performed a meta-analysis of fasting glucose levels in approximately 50,000 individuals, with replication in an additional 76,558 people and identified nine new genetic variants that influence fasting glucose levels. The investigators also found that several of these variants are also associated with increased risk of T2D. In a separate report, the MAGIC investigators led by Richard Watanabe studied blood sugar levels 2 hours after an oral glucose challenge. The authors found three new genetic variants that influence certain glucose levels, with one variant overlapping those variants found to be associated with T2D in the first study.
Author contacts:
Jose Florez (Massachusetts General Hospital, Boston, MA, USA) Author paper [5]
Tel: +1 617 643 3308; E-mail: [email protected]
Richard Watanabe (University of Southern California, Los Angeles, CA) Author paper [6]
Tel: +1 323 442 2053; E-mail: [email protected]
[7] Nature: Host factors implicated in influenza virus replication
DOI: 10.1038/nature08760
Hundreds of human factors that affect influenza virus replication have been identified, uncovering many new potential targets for antiviral design. The findings are reported in this week’s Nature.
Thomas Meyer and colleagues used a genome-wide RNA interference screen to systematically disable single genes in cultured human lung cells before infecting them with influenza virus. Some 287 genes were found to have a crucial role in influenza A virus replication, with many genes affecting the replication of other influenza viruses, including avian and pandemic swine-origin strains.
Blocking one target with a small molecule inhibitor reduced influenza virus replication by more than two orders of magnitude, and the study suggests that some targets may have broad-spectrum therapeutic potential against multiple influenza strains. The study also highlights the validity of targeting host rather than viral factors — existing drugs directed against viral targets all too often lose efficacy as the virus mutates and builds up resistance.
Author contact:
Thomas Meyer (Max Planck Institute for Infection Biology, Berlin, Germany)
Tel: +49 30 28 46 04 02; E-mail: [email protected]
[8] Chemical Biology: Finding psychiatric drugs in zebrafish?
DOI: 10.1038/nchembio.307
Zebrafish may provide a great platform for finding new drugs for neurological disorders suggest a paper published online in Nature Chemical Biology this week. The discovery in the 1950’s of drugs that act in the nervous system has been very important both for understanding neurobiology and treating neurological diseases. However, very few drugs have been discovered for these diseases since.
Randall Peterson, David Kokel and colleagues report that different types of neurological drugs caused distinct patterns of behaviours in zebrafish. By looking at the behavioral effects of a large number of chemicals, the investigators were able to identify new chemicals that affect behaviour and to predict how these chemicals might be acting.
This ‘behavioral barcoding’ of chemical effects in zebrafish provides a much needed new approach for identifying leads for neurological diseases.
Author contacts:
Randall Peterson, (Harvard Medical School, Charlestown, MA, USA)
Tel: +1 617 724 9569; E-mail: [email protected]
David Kokel (Harvard Medical School, Charlestown, MA, USA)
Tel: +1 617 726-6470; E-mail: [email protected]
[9] Geoscience: Indonesian earthquake risk
DOI: 10.1038/ngeo753
The September 2009 earthquake in Indonesia did not significantly relax the threat of a high-magnitude tsunami-generating earthquake in the region, reports a study online this week in Nature Geoscience. Stress on this segment of the fault has been accumulating for about 200 years, suggesting the need for urgent action
John McCloskey and colleagues investigated the stress released during the September 2009 earthquake of magnitude 7.6 near the Indonesian city of Padang, which is located on Sumatra near Siberut. They found that the quake did not rupture the section of the Sunda megathrust along the coast of Sumatra that had remained unbroken by a series of earthquakes after the 2004 quake and tsunami. The danger of strong shaking and the potential for tsunami generation as forecast earlier therefore is therefore still immanent.
Author contact:
John McCloskey (University of Ulster, UK)
Tel: +44 2870 324 769; E-mail: [email protected]
[10] Methods: Assembling the bigger picture
DOI 10.1038/nmeth.1416
A technique to bring the short sequence fragments – referred to as reads – generated by next generation sequencing technology to a length equivalent to traditional capillary sequencing reads, is published online this week in Nature Methods. It promises faster and more accurate genome assembly from short read data.
Next generation sequencing is far cheaper and provides much higher throughput than capillary-based sequencing, but it generates short sequence reads that are difficult to assemble into a long contiguous sequence. The quality of assembled genomes is therefore often not as good as that achievable with traditional sequencing.
Jay Shendure and colleagues assembled the genome of the human pathogen Pseudomonas aeruginosa, which has proven difficult to assemble previously and characterized a sample of the collective genomes of microorganisms from lake sediment. With far less sequencing effort they produced genomic information comparable to that obtained with capillary sequencing.
Author contact:
Jay Shendure (University of Washington, Seattle, WA, USA)
Tel: +1 206 685 8543; E-mail [email protected]
[11] And finally…Physics: ‘Chaos control’ spawns complex robotic behaviour
DOI 10.1038/nphy1508
Inspired by insects, a ‘smart’ six-legged robot that uses chaos control to react quickly to its environment can perform a variety of complex ‘cockroach-like’ behaviours. The hexapod is revealed in this week’s Nature Physics.
Mimicking even the simplest of animal behaviour, such as walking along uneven terrain, is a challenging task. But Poramate Manoonpong, Marc Timme, and colleagues show that by incorporating a simple but inherently chaotic pattern-generator into the control system of an autonomous robot enables it to exhibit adaptive behaviour that allows it to successfully navigate through a complex environment. The robot, which performs combinations of 11 basic behaviours, outstrips many of its rivals that operate with limited autonomy and perform just a few behaviours. The team designed the device, which uses 18 sensors to drive 18 motors by means of one simple neural circuit.
The result is a robot that swiftly changes gait to walk uphill, downhill or over rough terrain, whilst retaining the ability to avoid obstacles and react to light. And if it finds itself in a hole, the neural circuit operates chaotically, enabling the robot to free itself. So the hexapod quickly and reversibly adapts to new situations, but also retains the ability to learn over longer time scales.
Author contacts:
Poramate Manoonpong (Bernstein Center for Computational Neuroscience, Goettingen, Germany)
Tel: +49 551 3910 763; E-mail: [email protected]
Marc Timme (Max Planck Institute for Dynamics and Self-Organization, Goettingen, Germany)
Tel: +49 551 5176 440; E-mail: [email protected]
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Items from other Nature journals to be published online at the same time and with the same embargo:
Nature (http://www.nature.com/nature)
[12] DNMT1 maintains progenitor function in self-renewing somatic tissue
DOI: 10.1038/nature08683
NATURE BIOTECHNOLOGY (http://www.nature.com/naturebiotechnology)
[13] Enhanced antibody half-life improves in vivo activity
DOI: 10.1038/nbt.1601
[14] Rational design of cationic lipids for siRNA delivery
DOI: 10.1038/nbt.1602
NATURE CELL BIOLOGY (http://www.nature.com/naturecellbiology)
[15] Histone deacetylase and Cullin3–RENKCTD11 ubiquitin ligase interplay regulates Hedgehog signalling through Gli acetylation
DOI: 10.1038/ncb2013
[16] The Nup107‑160 complex and g‑TuRC regulate microtubule polymerization at kinetochores
DOI: 10.1038/ncb2016
[17] 53BP1-dependent robust localized KAP‑1 phosphorylation is essential for heterochromatic DNA double-strand break repair
DOI: 10.1038/ncb2017
[18] Cohesin cleavage and Cdk inhibition trigger formation of daughter nuclei
DOI: 10.1038/ncb2018
[19] Regulation of NF-kB inhibitor IkBa and viral replication by a KSHV microRNA
DOI: 10.1038/ncb2019
NATURE CHEMICAL BIOLOGY (http://www.nature.com/nchembio)
[20] Carbon metabolism–mediated myogenic differentiation
DOI: 10.1038/nchembio.301
[21] Differences in prion strain conformations result from non-native interactions in a nucleus
DOI: 10.1038/nchembio.306
NATURE CHEMISTRY (http://www.nature.com/nchem)
[22] Chemoenzymatic synthesis of differentially protected 3-deoxysugars
DOI: 10.1038/nchem.504
[23] Highly diastereoselective Csp3–Csp2 Negishi cross-coupling with 1,2-, 1,3- and 1,4-substituted cycloalkylzinc compounds
DOI: 10.1038/nchem.505
[24] Dianionic species with a bond consisting of two pentacoordinated silicon atoms
DOI: 10.1038/nchem.513
[25] Solution structure of a DNA double helix with consecutive metal-mediated base pairs
DOI: 10.1038/nchem.512
NATURE GENETICS (http://www.nature.com/naturegenetics)
[26] Somatic mutation of EZH2 (Y641) in follicular and diffuse large B-cell lymphomas of germinal center origin
DOI: 10.1038/ng.518
[27] AHI1 is required for outer segment development and is a modifier for retinal degeneration in nephronophthisis
DOI: 10.1038/ng.519
NATURE GEOSCIENCE (http://www.nature.com/ngeo)
[28] Contribution of Alaskan glaciers to sea-level rise derived from satellite imagery
DOI: 10.1038/ngeo737
[29] Slip-rate variability and distributed deformation in the Marmara Sea fault system
DOI: 10.1038/ngeo739
[30] The nature of millennial-scale climate variability during the past two glacial periods
DOI: 10.1038/ngeo740
[31] Vulnerability of deep groundwater in the Bengal Aquifer System to contamination by arsenic
DOI: 10.1038/ngeo750
NATURE IMMUNOLOGY (http://www.nature.com/natureimmunology)
[32] Regulation of hematopoietic stem cell differentiation by a single ubiquitin ligase-substrate complex
DOI: 10.1038/ni.1839
NATURE MATERIALS (http://www.nature.com/naturematerials)
[33] Many-body effects in electronic bandgaps of carbon nanotubes measured by scanning tunnelling spectroscopy
DOI: 10.1038/nmat2624
Nature MEDICINE (http://www.nature.com/naturemedicine)
[34] Epidermal injury and infection during poxvirus immunization is crucial for the generation of highly protective T cell–mediated immunity
DOI: 10.1038/nm.2078
[35] Notch-mediated expansion of human cord blood progenitor cells capable of rapid myeloid reconstitution
DOI: 10.1038/nm.2080
[36] Neutrophil elastase–mediated degradation of IRS-1 accelerates lung tumor growth
DOI: 10.1038/nm.2084
[37] Recognition of peptidoglycan from the microbiota by Nod1 enhances systemic innate immunity
DOI: 10.1038/nm.2087
NATURE METHODS (http://www.nature.com/nmeth)
[38] Bright cyan fluorescent protein variants identified by fluorescence lifetime screening
DOI: 10.1038/nmeth.1415
[39] FRT-seq: Amplification-free, strand-specific, transcriptome sequencing
DOI: 10.1038/nmeth.1417
NATURE NANOTECHNOLOGY (http://www.nature.com/nnano)
[40] Atomic structure of conducting nanofilaments in TiO2 resistive switching memory
DOI: 10.1038/nnano.2009.456
[41] Engineered biological nanofactories trigger quorum sensing response in targeted bacteria
DOI: 10.1038/nnano.2009.457
[42] Towards a quantum resistance standard based on epitaxial graphene
DOI: 10.1038/nnano.2009.474
Nature NEUROSCIENCE (http://www.nature.com/natureneuroscience)
[43] Glutamate co-release at GABA/glycinergic synapses is crucial for the refinement of an inhibitory map
DOI: 10.1038/nn.2478
[44] Synaptic activation of kainate receptors gates presynaptic CB1 signalling at GABAergic synapses
DOI: 10.1038/nn.2481
[45] Activation of subpopulations of excitatory neurons in the mammalian hindbrain or spinal cord evokes locomotion
DOI: 10.1038/nn.2482
NATURE PHOTONICS (http://www.nature.com/nphoton)
[46] Airy–Bessel wave packets as versatile linear light bullets
DOI: 10.1038/nphoton.2009.264
[47] Active magneto-plasmonics in hybrid metal–ferromagnet structures
DOI: 10.1038/nphoton.2009.265
[48] Ultrabroad-bandwidth arbitrary radio-frequency waveform generation with a silicon photonic chip-based spectral shaper
DOI: 10.1038/nphoton.2009.266
[49] Laser cooling of solids to cryogenic temperatures
DOI: 10.1038/nphoton.2009.269
Nature PHYSICS (http://www.nature.com/naturephysics)
[50] High-reflectivity high-resolution X-ray crystal optics with diamonds
DOI: 10.1038/nphys1506
[51] Evolution of spin excitations into the superconducting state in FeTe1-xSex
DOI: 10.1038/nphys1512
[52] Isolated optical vortex knots
DOI: 10.1038/nphys1504
Nature STRUCTURAL & MOLECULAR BIOLOGY (http://www.nature.com/natstructmolbiol)
[53] Structural basis for dsRNA recognition and interferon antagonism by Ebola VP35
DOI: 10.1038/nsmb.1765
*****************************************************
GEOGRAPHICAL LISTING OF AUTHORS
The following list of places refers to the whereabouts of authors on the papers numbered in this release. The listing may be for an author's main affiliation, or for a place where they are working temporarily. Please see the PDF of the paper for full details.
AUSTRALIA
Nedlands : 5
Perth : 5
Sydney: 5
BANGLADESH
Dhaka: 31
BELARUS
Grodno: 47
CANADA:
Burnaby: 14
Calgary: 20
Montreal: 5, 6, 27
Ottawa: 2, 5
Prince George: 28
Toronto: 3
Vancouver: 2, 8, 14, 26, 28
CHINA
Beijing: 4
Hubei: 19
CROATIA
Split: 5
Zagreb: 5
DENMARK
Aarhus: 5, 6
Glostrup: 5, 6
Copenhagen: 5, 6
Gentofte: 5, 6
Odense: 5, 6
FINLAND
Helsinki: 5, 6
Jakobstad: 5, 6
Kuopio: 5, 6
Oulu: 5
Seinajoki: 5
Turku: 5
FRANCE
Bobigny: 5
Bordeaux: 44
Cachan: 33
Chatillon: 33
Essonnes: 5, 6
Evry: 5
Lille: 5, 6
Nancy: 5
Orsay: 6
Paris: 5, 6, 33
Poitiers: 5
Saint-Louis: 49
Toulouse: 28
Villejuif: 5
GERMANY
Aachen: 7
Berlin: 7, 22, 47
Bonn: 3
Darmstadt: 17
Dresden : 5, 6
Düsseldorf: 5, 6
Goettingen: 11
Hannover: 11, 27
Heidelberg: 27
Karlsruhe: 29
Konstanz: 47
Leipzig: 5, 6
Mannheim: 3
Munich: 3, 5, 6, 23
Munster: 5, 24
Neuherberg: 5, 6
Nuthetal: 5
Potsdam: 22, 29, 30
Regensburg: 23
Wurzburg: 7
GREECE
Athens: 5
Mytilene: 30
ICELAND
Kopavogur: 5
Reykjavik: 5, 6
INDONESIA
Bandung: 9
ITALY
Bolzano: 5
Cagliari: 5
Chieti-Pescara: 27
Genova: 27
Naples: 5, 15
Messina : 27
Milan: 5, 42
Pisa: 49
Pomezia: 15
Pozzilli: 15
Rome: 15, 27
Verona: 3
JAPAN
Aichi: 1
Chiba: 17
Hyogo: 21
Okazaki: 24
Osaka: 1
Saitama: 21
Tokyo: 1, 24
NETHERLANDS
Amsterdam: 5, 38
Leiden: 5
Nijmegen: 27
Rotterdam: 5, 6
NORWAY
Oslo: 27
PORTUGAL
Coimbra: 44
SOUTH KOREA
Chungju: 19
Seoul: 40, 43
Suwon: 40
SPAIN
Madrid: 5, 47
Raras: 27
SWEDEN
Gothenburg: 42
Linkoping: 42
Malmo: 5, 6
Stockholm: 5, 6, 45
Umea: 5
Uppsala: 5, 6
SWITZERLAND
Lausanne: 3, 5, 6
Zurich: 22, 25
UNITED KINGDOM
Belfast: 5
Birmingham: 17
Bristol: 5, 52
Cambridge: 5, 6, 9, 30, 39
Coleraine: 9
Didcot: 51
Dundee: 5, 6
Edinburgh: 5, 6, 9
Essex: 39
Exeter: 5, 6
Glasgow: 52
Lancaster: 42
Leeds: 30
Liverpool: 9
London: 3, 5, 6, 30, 31
Newcastle: 6
Norwich: 6
Oxford: 5, 6, 18
Southampton: 5, 6, 52
Sussex: 17
Teddington: 42
UNITED STATES OF AMERICA
Alabama
Birmingham: 36
Arizona
Flagstaff: 28
California
Berkeley: 53
La Jolla: 27
Los Angeles: 5, 6, 27
Palo Alto: 12
San Diego: 13, 26
San Francisco: 5, 8, 21, 43
Stanford: 12
Colorado
Denver: 31
Delaware
Newark: 31
Florida
Orlando: 46
Illinois
Argonne: 50, 51
Evanston: 48
Indiana
West Lafayette: 48
Iowa
Ames: 53
Maine
Bar Harbor: 13
Maryland
Baltimore: 5, 6
Bethesda: 3, 5, 6, 16
College Park: 41
Massachusetts
Boston: 5, 6, 8, 20, 34, 36
Cambridge: 5, 6, 8, 14, 18, 20, 47
Charlestown: 8
Framingham: 5, 6
Michigan
Ann Arbor: 5, 6, 27
Minnesota
Minneapolis: 5, 6
Rochester: 6
Missouri
St Louis: 5, 32
New Mexico
Albuquerque: 49
New York
Ithaca: 46
New York: 32, 53
Upton: 50
North Carolina
Chapel Hill: 5, 6
Pennsylvania
King of Prussia: 5, 6
Philadelphia: 4, 37
Pittsburgh: 36, 43
Tennessee
Knoxville: 51
Oak Ridge: 51
Texas
Dallas: 5, 16, 53
Houston: 5, 6
San Antonio: 19
Virginia
Reston: 31
Washington
Seattle: 5, 6, 10, 35
PRESS CONTACTS…
For media inquiries relating to embargo policy for all the Nature Research Journals:
Rachel Twinn (Nature London)
Tel: +44 20 7843 4658; E-mail: [email protected]
Neda Afsarmanesh (Nature New York)
Tel: +1 212 726 9231; E-mail: [email protected]
Ruth Francis (Head of Press, Nature, London)
Tel: +44 20 7843 4562; E-mail: [email protected]
For media inquiries relating to editorial content/policy for the Nature Research Journals, please contact the journals individually:
Nature Biotechnology (New York)
Michael Francisco
Tel: +1 212 726 9288; E-mail: [email protected]
Nature Cell Biology (London)
Sowmya Swaminathan
Tel: +44 20 7843 4656; E-mail: [email protected]
Nature Chemical Biology (Boston)
Andrea Garvey
Tel: +1 617 475 9241, E-mail: [email protected]
Nature Chemistry (London)
Stuart Cantrill
Tel: +44 20 7014 4018; E-mail: [email protected]
Nature Genetics (New York)
Myles Axton
Tel: +1 212 726 9324; E-mail: [email protected]
Nature Geoscience (London)
Heike Langenberg
Tel: +44 20 7843 4042; E-mail: [email protected]
Nature Immunology (New York)
Laurie Dempsey
Tel: +1 212 726 9372; E-mail: [email protected]
Nature Materials (London)
Vincent Dusastre
Tel: +44 20 7843 4531; E-mail: [email protected]
Nature Medicine (New York)
Juan Carlos Lopez
Tel: +1 212 726 9325; E-mail: [email protected]
Nature Methods (New York)
Hugh Ash
Tel: +1 212 726 9627; E-mail: [email protected]
Nature Nanotechnology (London)
Peter Rodgers
Tel: +44 20 7014 4019; Email: [email protected]
Nature Neuroscience (New York)
Kalyani Narasimhan
Tel: +1 212 726 9319; E-mail: [email protected]
Nature Photonics (Tokyo)
Oliver Graydon
Tel: +81 3 3267 8776; E-mail: [email protected]
Nature Physics (London)
Alison Wright
Tel: +44 20 7843 4555; E-mail: [email protected]
Nature Structural & Molecular Biology (New York)
Sabbi Lall
Tel: +1 212 726 9326; E-mail: [email protected]
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